Proteomics Identifies Substrates and a Novel Component in hSnd2-Dependent ER Protein Targeting

Publication date

2022-09-19

Authors

Tirincsi, Andrea
O’Keefe, Sarah
Nguyen, Duy
Sicking, Mark
Dudek, Johanna
Forster, FriedrichORCID 0000-0002-6044-2746ISNI 0000000017448240
Jung, Martin
Hadzibeganovic, Drazena
Helms, Volkhard
High, Stephen

Editors

Advisors

Supervisors

Document Type

Article
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cc_by

Abstract

Importing proteins into the endoplasmic reticulum (ER) is essential for about 30% of the human proteome. It involves the targeting of precursor proteins to the ER and their insertion into or translocation across the ER membrane. Furthermore, it relies on signals in the precursor polypeptides and components, which read the signals and facilitate their targeting to a protein-conducting channel in the ER membrane, the Sec61 complex. Compared to the SRP- and TRC-dependent pathways, little is known about the SRP-independent/SND pathway. Our aim was to identify additional components and characterize the client spectrum of the human SND pathway. The established strategy of combining the depletion of the central hSnd2 component from HeLa cells with proteomic and differential protein abundance analysis was used. The SRP and TRC targeting pathways were analyzed in comparison. TMEM109 was characterized as hSnd3. Unlike SRP but similar to TRC, the SND clients are predominantly membrane proteins with N-terminal, central, or C-terminal targeting signals.

Keywords

differential protein abundance analysis, endoplasmic reticulum, guided entry of tail-anchored proteins, membrane proteins, protein targeting, protein translocation, Sec61 complex, signal recognition particle, SRP-independent targeting, General Biochemistry,Genetics and Molecular Biology

Citation

Tirincsi, A, O’Keefe, S, Nguyen, D, Sicking, M, Dudek, J, Förster, F, Jung, M, Hadzibeganovic, D, Helms, V, High, S, Zimmermann, R & Lang, S 2022, 'Proteomics Identifies Substrates and a Novel Component in hSnd2-Dependent ER Protein Targeting', Cells, vol. 11, no. 18, 2925. https://doi.org/10.3390/cells11182925