Severe hypertriglyceridaemia and pancreatitis in a patient with lipoprotein lipase deficiency based on mutations in lipoprotein lipase (LPL) and apolipoprotein A5 (APOA5) genes
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2019-04-01
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taverne
Abstract
A 44-year-old woman was admitted with pancreatitis caused by hypertriglyceridaemia (fasting triglycerides 28 mmol/L). She used oral contraceptives and ezetimibe 10 mg. She was overweight (body mass index 29.7 kg/m 2). Diabetes mellitus was ruled out, as were nephrotic syndrome, alcohol abuse, hypothyroidism and dysbetalipoproteinaemia. Genetic analysis revealed mutations in two genes involved in triglyceride metabolism (apolipoprotein A5 and lipoprotein lipase [LPL]). The LPL activity was 45% compared with pooled healthy controls. The post-heparin triglyceride reduction was 6%, compared with a normal reduction of >20%. The patient was initially treated with gemfibrozil, but this was discontinued due to side effects. Dietary triglyceride restriction and discontinuation of the oral contraceptives lowered the plasma triglycerides within 2 weeks to 3.4 mmol/L. Hypertriglyceridaemia is a risk factor for pancreatitis and cardiovascular disease, and has a broad differential diagnosis including genetic causes. Patients can achieve near-normal triglyceride values with a low-fat diet only.
Keywords
cardiovascular medicine, genetics, lipid disorders, pancreatitis, Humans, Lipoprotein Lipase/deficiency, Hyperlipoproteinemia Type I/genetics, Adult, Female, Apolipoprotein A-V/genetics, Mutation, Pancreatitis/genetics, Hypertriglyceridemia/genetics, Taverne, General Medicine, Case Reports, Journal Article
Citation
Koopal, C, Bemelmans, R, Marais, A D & Visseren, F L J 2019, 'Severe hypertriglyceridaemia and pancreatitis in a patient with lipoprotein lipase deficiency based on mutations in lipoprotein lipase (LPL) and apolipoprotein A5 (APOA5) genes', BMJ Case Reports, vol. 12, no. 4, e228199. https://doi.org/10.1136/bcr-2018-228199